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Carbidopa blocks the peripheral metabolism of levodopa to dopamine, decreasing the peripheral adverse effects of levodopa, such as nausea and vomiting, while increasing levodopa’s availability in the brain.

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Levodopa, combined with a peripheral decarboxylase inhibitor (PDI) such as carbidopa, is the criterion standard of symptomatic treatment for PD; it provides the greatest antiparkinsonian efficacy in moderate to advanced disease with the fewest acute adverse effects.

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Researchers also hope that discovering genes will help identify new ways of treating PD.

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Multifocal Dystonia: affects two or more distant regions of the body, such as the upper face and the hand.

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For people with advanced Parkinson disease whose symptoms can no longer be controlled by medication, surgical treatment with deep brain stimulation may be an option.

Other possible causes include abnormal development of the brain, prenatal factors that directly or indirectly damage neurons in the developing brain, premature birth, and brain injuries that occur in the first few years of life.

The National Eye Institute (NEI) supports work on the study of blepharospasm and related problems, and the Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD) supports work on dystonia, including the rehabilitation for the disorder.

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This combination provides the greatest symptomatic benefit with the fewest short-term adverse effects.

PSP is often misdiagnosed because some of its symptoms are much like those of PD, Alzheimer’s disease, and other brain disorders.

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